J3-4108 — Final report
1.
Suppression of membrane vesiculation as anticoagulant and anti-metastatic mechanism

Mechanism that was previously observed in systems of artificial membranes was proved in red blood cells. We found that stability of narrow nech connecting the bud and the cell is important in suppression of microvesiculation.

COBISS.SI-ID: 4516459
2.
Extracellular vesicle-mediated transfer of membranous components from the highly malignant T24 urinary carcinoma cell line to the non-malignant RT4 urinary papilloma cell line

Extracellular vesicles from the medium od cell line T24, marked wih nonspecific fluorescent marker DiO and B subunit of choleratoxin incorporated into urothelial papilloma cells RT4

COBISS.SI-ID: 10148948
3.
A 32-month follow-up study of nanovesicle concentrations in blood from 12 patients with gastrointestinal stromal tumour treated with imatinib

We followed concentration of nanovesicles in isolates from blood of patients before and dzring the treatment and found dramatic increase of concentration in the first week of treatment while the concentrations were dropped to the level of healthy control in the long term

COBISS.SI-ID: 9629012
4.
Vesiculation of biological membrane driven by curvature induced frustrations in membrane orientational ordering

Anisotropic constituents attain orientational ordering in the curvature field. In closed membranes this induces frustrations in the order parameter while for a given shape of constituents the frustration distribution drives vesiculation of the membrane

COBISS.SI-ID: 4516971
5.
Stability of membranous nanostructures. A possible key mechanism in cancer progression

Stability of membranous nanostructures is considered as a mechanism in cancer development. Microvesicles and nanotubes are involved in metastasis and tumor growth.

COBISS.SI-ID: 4415339